Department hypertensive nephropathy original small arteries cause hypertension benign nephrosclerosis. (Called renal hypertensive arteriosclerosis) and malignant arteriolar nephrosclerosis malignoYacompa?ado of appropriate clinical disease usually many years history of hypertension disease. Tubular damage earlier than glomerular. Urine night increases. Function solta urine concentration, unless the changes urinate, there is less proteinuria, hematuria and low mirror tube, normally there are other target organ disease.
The kidney is made through the capillaries surrounding organs, to filter the toxin body. Mintra prevents protein and blood cells so oozes artery. Hypertension Blood pressure demand increased intravascular, send pale leak, but pale leak can filter system of kidney damage creates vicious circle. The long period may cause serious injury, kidney can increase compensatory end anticipated exhaustion. This because there are not terrible hematuria, proteinuria but there is very terrible.
Corrientes causes of disease are: The incidence of kidney blood pressure wounds, severe level of blood pressure and time are then positive relationship. other possible factors include gender, race, diabetes, hyperlipidemia and hyperuricemia, these factors include each other even more hurt in kidney. men more than women, think of that kidney is injured. The age of onset of primary hypertension is usually 25-45 years, but the kidney damage caused by hypertension age of onset of clinical symptoms is usually 40-60 years. The earliest symptoms is that bedwetting increases ischemic tubular Checking pathological changes occurred, condensed empeza lower urinary function. appears after proteinuria, glomerular Checking has occurred pathological changes. The level of proteinuria is usually mild to moderate (+ or ++), in 24 hours, the amount of urinary protein generally does not exceed 20g, but are very few in number proteinuria patient. If you microscopic examination of urine sediment, you can find red blood cell and tube are little, a few patients because of the glomerular capillary cracks, passenger gross hematuria occurs. Studies have shown that in patients with primary hypertension, there millionth pacietes can become kidney failure, hypertensive untreated patients with significantly impaired renal function compared to those without hypertension.
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